Venous and Arterial Blood Lactate in HIV-Infected Patients

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dc.contributor.authorZala, Carlos
dc.contributor.authorHarris, Marianne
dc.contributor.authorOchoa, Claudia
dc.contributor.authorTocci, Mariel
dc.contributor.authorQuercia, Romina
dc.contributor.authorMittelman, Graciela
dc.contributor.authorGraciela, Héctor
dc.contributor.authorCahn, Pedro
dc.contributor.authorMontaner, Julio SG
dc.date.accessioned2024-05-23T23:49:29Z
dc.date.available2024-05-23T23:49:29Z
dc.date.issued2002-12
dc.descriptionFil: Zala C. Fundación Huésped, Buenos Aires; Argentinaes_ES
dc.descriptionFil: Ochoa C. Fundación Huésped, Buenos Aires; Argentinaes_ES
dc.descriptionFil: Cahn P. Fundación Huésped, Buenos Aires; Argentinaes_ES
dc.description.abstractHyperlactataemia is increasingly recognised as a complication of nucleoside analogue-based antiretroviral therapy [1–5]. There is, however, some debate about the accuracy of peripheral venous blood lactate levels and whether these adequately reflect arterial lactate levels. This correlation has been demonstrated in the non-HIV, emergency medicine literature [6,7], but has not been studied in HIV patients receiving antiretroviral therapy. Therefore, we undertook the present study to compare venous versus arterial lactate levels in HIV patients who had consistently high random venous lactate while receiving chronic stavudine-based antiretroviral therapy.
dc.formatapplication/pdfes_ES
dc.identifier.doihttps://doi.org/10.1177/135965350200700412
dc.identifier.urihttps://repositorio.huesped.org.ar/handle/123456789/1361
dc.languageENGes_ES
dc.provenancePublishedes_ES
dc.relation.ispartofseriesAntiviral Therapy;2002;7(4):309-310
dc.rightsopenAccesses_ES
dc.subjectHIVes_ES
dc.titleVenous and Arterial Blood Lactate in HIV-Infected Patientses_ES
dc.typeArticuloes_ES

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