A dynamic interplay of circulating extracellular vesicles and galectin-1 reprograms viral latency during HIV-1 infection

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dc.contributor.authorRubione, Julieta
dc.contributor.authorDuette, Gabriela
dc.contributor.authorPerez, Paula
dc.contributor.authorPereyra Gerber, Pablo
dc.contributor.authorSalido, Jorge
dc.contributor.authorCagnoni, Ana
dc.contributor.authorGuzman, Laura
dc.contributor.authorAdamczyk, Ariel
dc.contributor.authorSued, Omar
dc.contributor.authorGhiglione, Yanina
dc.contributor.authorLaufer, Natalia
dc.contributor.authorMariƱo, Karina
dc.contributor.authorRabinovich, Gabriel
dc.contributor.authorOstrowski, Mario
dc.date.accessioned2024-05-21T13:37:38Z
dc.date.available2024-05-21T13:37:38Z
dc.date.issued2019
dc.description.abstractHIV-positive individuals on antiretroviral therapy(art) have detectable cell-associated unspliced (ca-Us) HIV rNain cD4+ t-cells from blood which varies with time. additionally, werecently showed that circadian transcription factors, circadian Loco-motor output cycles Kaput (cLocK) and brain and Muscle arnt-likeprotein-1 (bMaL1), bind to the HIV Ltr and increase HIV transcrip-tion. We hypothesised that circadian rhythms exert transcriptionalcontrol on latent HIV.
dc.identifier.citationJournal of Virus Eradication 5:10-11. DOI: 10.1016/S2055-6640(20)31039-6
dc.identifier.other10.1016/S2055-6640(20)31039-6
dc.identifier.urihttps://repositorio.huesped.org.ar/handle/123456789/1007
dc.relation.ispartofseriesJournal of Virus Eradication; 5
dc.subjectHIV 1
dc.subjectpost-cART initiation
dc.titleA dynamic interplay of circulating extracellular vesicles and galectin-1 reprograms viral latency during HIV-1 infection

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